例句與造句

1. Deficiency of Factor XIII leads to defective cross-linking of fibrin and vulnerability to late re-bleeds when the primary hemostatic plug is overwhelmed.
2. Factor XIII is a transglutaminase that circulates in the plasma as a heterotetramer of two catalytic A subunits and two carrier B subunits.
3. Transglutaminases were first described in 1959 . The exact biochemical activity of transglutaminases was discovered in blood coagulation protein factor XIII in 1968.
4. Besides aiding the overall understanding of clotting, his discoveries helped researchers develop treatments for people who have bleeding problems because they lack Factor XIII.
5. During blood clotting, the fibronectin remains associated with the clot, covalently cross-linked to fibrin with the help of Factor XIII ( fibrin-stabilizing factor ).
6. It's difficult to find factor xiii in a sentence. 用factor xiii造句挺難的
7. A substance they identified, initially named Laki-Lorand factor, became known as factor XIII . Laki was awarded an honorary medical degree from the University of Debrecen.
8. Deficiencies in other factors, such as factor XIII or factor VII are occasionally seen, but may not be associated with severe bleeding and are not as commonly diagnosed.
9. As the enzyme is quite specific for monocytes and macrophages, determination of the presence of factor XIII may be used to identify and classify malignant diseases involving these cells.
10. In the presence of calcium the carrier subunits dissociate from the catalytic subunits, leading to a 3D change in conformation of factor XIII and hence the exposure of catalytic cysteine residue.
11. Effects on other clotting factors : Unlike thrombin, ancrod does not directly activate Factor XIII, nor does it produce platelet aggregation nor cause the release of ATP, potassium, or serotonin from platelets.
12. Factor XIII deficiency is classified into two categories : type I deficiency, characterized by the lack of both the A and B subunits; and type II deficiency, characterized by the lack of the A subunit alone.
13. The introduction of rFXIII as a treatment for factor XIII deficiency eliminates the risk of pathogenic infection present in plasma-based treatments . rFXIII treatment would also not be dependent on blood donations, consequently increasing availability and product quality.
14. As Factor XIII is composed of two subunit protein, A and B, for which the genes are located on different chromosomes, administration of recombinant A subunit improves clot stability and is becoming a therapeutic option for patients with this condition.
15. Thrombin is a central enzyme in the coagulation process : it generates fibrin from fibrinogen, and activates a number of other enzymes and cofactors ( factor XIII, factor XI, factor V and factor VIII, TAFI ) that enhance the fibrin clot.
16. Ribosomal protein S19 has been shown to Coagulation factor XIII ) is also known to bind and probably inhibit Macrophage migration inhibitory factor; though S19 oligomers themselves share MCIP's function as another very strong macrophage chemoattractant and bind to anaphylotoxin C5 receptor
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