Recently , research on the sos3 / athktl double mutants in arabidopsis showed functional disruption of athktl was shown to suppress t he salt sensitive phenotype of sos3 mutant indicating that wild - type sos3 may inhibit the activity of athktl as a na + influx transporter . but hitherto there is no evidences in electrophysiology about regulation of k + and na + influx by sos3 protein 結(jié)果暗示athkt1可能是一個(gè)na ~ +內(nèi)流轉(zhuǎn)運(yùn)體并起了調(diào)節(jié)k ~ + 、 na ~ +吸收系統(tǒng)的作用,并且野生型sos3蛋白可能抑制athkt1蛋白na ~ +內(nèi)流轉(zhuǎn)運(yùn)體的活性, athkt1可能是sos途徑的調(diào)節(jié)對(duì)象之一。